Red Ox+ is a nutritional supplement composed of a special blend of antioxidants and vitamins, designed to reduce free radicals and the damage they cause. This blend was formulated by doctors who used years of published research to create the optimal blend for a person with diabetes.
Red Ox+ should be taken once daily
. It works best when taken with meals. paired with a healthy diet and should be added to your current treatment plan under medical supervision. It is not a cure for diabetes but helps your body to eliminate unhealthy free radicals and reduce their damaging side effects.
Red Ox+ provides nutritional support to help:
- Increase Wound Healing
- Decrease Bacterial Burden in Wounds
- Decrease Antibiotic Resistance
- Increase Nitric Oxide Production
- Produces More Collagen in Wounds
- Promotes Strong Bone Health
- Ameliorate Peripheral Neuropathy
- Promote Sweat Gland Function
Red Ox+ also helps with:
Osteoporosis, fractures, tendons, diabetes, athletic recovery, heart and vascular health, skin/nail/hair/sweat gland function, mental focus/anti-depression/autism, Concussion, Preoperative and postoperative healing.
Preventing oxidative damage is paramount to aid proper bone and wound healing. Oxidative damage also results in skin and muscle flap and transfer failures. Therefore, all pre and post operative patients should get proper antioxidant supplements through Red Ox. Patients with any type of wound or fracture or poor skin and bone stock (osteopenia) should also get Red Ox.
More Specifics on Bone Health, Wound Healing, and Pre/Post Operative Healing (particularly muscle flaps):
Oxidative stress will negatively impact bone remodeling. Bone remodeling is a complex cycle, which lasts approximately 6 months, and harbors three main cells (osteoclasts, osteoblasts, and osteocytes) which, under normal circumstances will be harmoniously regulated. Osteoclasts lay down new bone while osteoclasts resorb bone. Oxidative stress activates the differentiation of pre-osteoclasts to adult osteoclasts while inducing cell death of osteoblasts and osteocytes and augmenting bone resorption (thus stopping bone from forming). Recent literature has displayed that osteocytes constitute 90% of the bone cell population and are essential for bone remodeling. Oxidative stress causes osteocyte death.
Oxidative stress will inhibit wound remodeling. Current research shows that wounds exposed to the slightest increase in oxidative stress will have increased numbers of inflammatory mediators in the wound bed which will inhibit re-epithelialization, inhibit collagen/fibroblast formation, and favor a rich biofilm which may harbor anaerobic bacteria not found in culture. Matrix metalloproteinases (MMPs) are also increased during oxidative stress and are capable of degrading extracellular matrix proteins, releasing cell death ligands resulting in chronic wounds.
Skin and Muscle Flaps
Numerous studies have shown that oxidative stress can cause inflammatory changes causing decreased blood supply resulting in flap necrosis. The increase in oxidative stress can cause ischemia reperfusion injury (I/R). Necrosis to the distal region of random flaps is a major problem. I/R occurs when the reperfusion creates and harbors oxygen-derived free radicals which are deleterious to the tissues. Flaps in diabetics present their own risk as oxidative stress, is already significantly elevated in this population and diminishes the microvascular supply resulting in necrosis. Supplementation with antioxidants will also increase vascular endothelial growth factor increasing blood supply to the flap.